36374 Results for: "Lead+acetate+solution"

Supplier: Apollo Scientific

Glyoxal dimethyl acetal 60% in aqueous solution

Supplier: VWR Chemicals

Dose for preparation of 1 litre of solution of concentration 0.1 mol/l

Supplier: VWR Chemicals

Ready to use test solution (TS).

Supplier: Thermo Fisher Scientific

Lead(II) acetate trihydrate 99+% ACS

Supplier: G-Biosciences

Lithium acetate 1 mol/l in aqueous solution

Supplier: VWR Chemicals

EDTA disodium salt concentrate for dilution to 0.01 mol in aqueous solution, ConvoL NORMADOSE® volumetric solution

Supplier: VWR Chemicals

Iodine monobromide solution 20 g/l in acetic acid (99%) Reag. Ph. Eur. 1045901

Supplier: Thermo Fisher Scientific

Glyoxal dimethyl acetal 60% in aqueous solution

Supplier: Roth Carl

Lead(II) acetate trihydrate

Supplier: VWR Chemicals

EDTA disodium salt 0.05 mol/l (0.1 N) in aqueous solution, AVS TITRINORM volumetric solution

Supplier: VWR Chemicals

Perchloric acid 0.1 mol/l (0.1 N) in anhydrous acetic acid, AVS TITRINORM Reag. Ph. Eur. volumetric solution

Supplier: Bioss

The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.

Supplier: Bioss

The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.

Supplier: Merck

Lead(II) acetate trihydrate, Sigma-Aldrich®

Supplier: Thermo Fisher Scientific

Lead(II) acetate trihydrate 99.0-103.0% ACS

Supplier: Bioss

The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.

Supplier: Bioss

The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.

Supplier: VWR Chemicals

EDTA disodium salt 7,444 g/l in aqueous solution, AVS TITRINORM

Supplier: Bioss

The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.

Supplier: VWR Chemicals

Sodium acetate buffer, VWR®, 3 M, pH value: 5,2

Supplier: VWR Chemicals

Contents: ammonium acetate R / water R / hydrochloric acid R1.

Supplier: SIGMA ALDRICH MICROSCOPY

Alcianblue 8GX solution has been used as a staining dye for chondrocytic, osteoblastic and adipocytic differentiation. It has also been used for the staining of sections in immunohistochemistry.

Supplier: VWR Chemicals

A 4 g/l solution in a mixture of 5 volumes of anhydrous acetic acid R and 95 volumes of butanol R.

Supplier: G-Biosciences

Cloning Buffer, Sodium acetate buffer solution pH 5,2 (3 mol/l)

Supplier: PanReac AppliChem

Used for the purification of oligonucleotides.

Supplier: VWR Chemicals

Ninhydrin 0.2% in 1-butanol : acetic acid (95:5 v:v)

Supplier: MP Biomedicals

Lead tetraacetate smoothly induces the addition of difluorodiiodomethane to alkenes and alkynes.

Supplier: Thermo Fisher Scientific

Cloning Buffer, Sodium acetate buffer solution pH 5,2 (3 mol/l)

Supplier: VWR Chemicals

1ml = 5.6° dH/100 H₂O

Supplier: SIGMA ALDRICH MICROSCOPY

Bouin's solution is an excellent fixative for preserving soft and delicate structures. It is prepared with saturated picric acid, formaldehyde and acetic acid.