215 Results for: "2-Deoxy-D-glucose, [1-3H]&amp"

Supplier: MORAVEK BIOCHEMICALS MS

2-Deoxy-D-glucose, [1-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

D-Glucose, [2-3H(N)] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

D-Glucose, [2-3H(N)] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

5'-Deoxy-5-fluorouridine, [6-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

2'-Deoxy-2'-fluorouridine, [5'-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

3'-Deoxy-3'-fluorothymidine, [methyl-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

2'-Deoxy-2'-fluoroarabinofuranosylcytosine, [3H] ≥97% (by HPLC), MORpure™

Supplier: Ambeed

4-Amino-1-((2R,3S,4R,5R)-3-fluoro-4-hydroxy-5-(hydroxymethyl)tetrahydrofuran-2-yl)pyrimidin-2(1H)-one, Purity: 95%, CAS number: 56632-83-8, Appearance: White to Yellow Solid, Storage: Keep in dark place, Inert atmosphere, Room temperature, Size: 100MG

Supplier: MORAVEK BIOCHEMICALS MS

1-(2-Deoxy-2-fluoroarabinofuranosyl)-uracil, [5-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

3'-Deoxy-2',3'-didehydrothymidine, [methyl-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

Deoxy(2')adenosine-5'-triphosphate, tetraammonium salt, [2,8-3H] ≥95% (by HPLC)

Supplier: LGC Standards

TRC 3-(2-Deoxy-Beta-D-erythro-pentofuranosyl)pyrimido[1,2-a]purin-10(3H)-one

Supplier: LGC Standards

TRC 3-(2-Deoxy-beta-D-erythro-pentofuranosyl)pyrimido[1,2-a]purin-10(3H)-one-13C3

Supplier: MORAVEK BIOCHEMICALS MS

5-Hydroxymethyl-2'-deoxyuridine, [3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

5-Aza-2'-deoxycytidine, [6-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

5-Iodo-2'-deoxyuridine, [6-3H] ≥97% (by HPLC), MORpure™

Supplier: MORAVEK BIOCHEMICALS MS

2'-Deoxyuridine-5'-monophosphate, diammonium salt, [5-3H] ≥96% (by HPLC)

Supplier: MORAVEK BIOCHEMICALS MS

2'-Deoxyuridine-5'-triphosphate, tetraammonium salt, [5-3H] ≥96% (by HPLC)

Supplier: Bioss

Hydrolyzes UDP-glucose to glucose 1-phosphate and UMP and ADP-ribose to ribose 5-phosphate and AMP. The physiological substrate is probably UDP-glucose. Poor activity on other substrates such as ADP-glucose, CDP-glucose, GDP-glucose and GDP-mannose.

Supplier: Adipogen

Mitochondria-derived peptide (MDP). Promotes biosynthesis of the endogenous AMP analog AICAR and consequently AMP-activated protein kinase (AMPK). Induces cellular and systemic glucose uptake and improves insulin sensitivity. Shown to prevent diet-induced obesity (DIO) in mice. Potential anti-obesity and anti-aging compound.

Supplier: Adipogen

CTRP2 (C1qTNF-related protein 2) belongs to a highly conserved family of adiponectin paralogs. CTRP2 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism.

Supplier: Adipogen

CTRP6 belongs to a highly conserved family of adiponectin paralogs. CTRP6 mediates activation of AMP-activated protein kinase (AMPK) in skeletal muscle cells, thereby regulating glucose and lipid metabolism. Mediates the phosphorylation of acetyl-CoA carboxylase (ACC).

Supplier: MilliporeSigma

An antihyperglycemic agent that lowers blood glucose levels without stimulating insulin secretion, promoting weight gain or causing hypoglycemia. Reported to stimulate AMP-activated protein kinase (AMPK) leading to the reduction in acetyl-CoA carboxylase (ACC) activity and induction of fatty acid oxidation.

Supplier: Adipogen

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Prosci

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Adipogen

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Prosci

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Adipogen

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Adipogen

CTRP5 (C1qTNF-related protein 5; C1QTNF5) belongs to a highly conserved family of adiponectin paralogs. CTRP5 mediates activation of AMP-activated protein kinase (AMPK) in muscle and liver cells, thereby regulating glucose and lipid metabolism. Serum levels of CTRP5 are significantly higher in obese/diabetic animal models compared to normal controls. Furthermore, CTRP5 may be a putative biomarker for mitochondrial dysfunction. Defects in C1QTNF5 are a cause of late-onset retinal degeneration (LORD).

Supplier: Rockland Immunochemical

Cyclic AMP-dependent transcription factor ATF-4 (ATF4) is a basic leucine-zipper (bZip) transcription factor, which regulates amino acid metabolism, DNA damage repair, chromatin remodeling, and apoptosis in response to cellular and ER stress. ATF4 works with various proteins, such as C/EBP homology protein (CHOP), aspargine synthetase (ASNS), and cAMP response element (CRE) among others to mediate cellular stress. ATF4 also regulates glucose homeostasis by suppressing beta-cell proliferation and insulin production. Furthermore, ATF4 targets the histone demethylase JMJD3 to alter chromatin structure and enhance gene transcription in response to amino acid deprivation. Anti-ATF4 is ideal for researchers interested in Cell Signaling, Oncology, Cell Differentiation, and Apoptosis; relevant pathways include MAPK signaling pathways, Activation of cAMP-Dependent PKA, CREB pathways, GPCR pathways and Rho Family GTPases.