12756 Results for: "Water+Baths,+Unstirred&pageNo=48"

Supplier: Bioss

Transmembrane adapter protein which associates with KLRK1 to form an activation receptor KLRK1-HCST in lymphoid and myeloid cells; this receptor plays a major role in triggering cytotoxicity against target cells expressing cell surface ligands such as MHC class I chain-related MICA and MICB, and UL16-binding proteins (ULBPs); these ligands are up-regulated by stress conditions and pathological state such as viral infection and tumor transformation. Functions as docking site for PI3-kinase PIK3R1 and GRB2. Interaction of ULBPs with KLRK1-HCST triggers calcium mobilisation and activation of the PIK3R1, MAP2K/ERK, and JAK2/STAT5 signaling pathways. Both PIK3R1 and GRB2 are required for full KLRK1-HCST-mediated activation and ultimate killing of target cells. In NK cells, KLRK1-HCST signaling directly induces cytotoxicity and enhances cytokine production initiated via DAP12/TYROBP-associated receptors. In T-cells, it provides primarily costimulation for TCR-induced signals. KLRK1-HCST receptor plays a role in immune surveillance against tumors and is required for cytolysis of tumors cells; indeed, melanoma cells that do not express KLRK1 ligands escape from immune surveillance mediated by NK cells.

Supplier: EJENDALS

INSOLE JALAS FX2 SUPREME EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

BOOT JALAS 2118 VIP SAFETY EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 7138 ZENIT EVO EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

BOOT JALAS 1378 HEAVY DUTY EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 1538 TERRA EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 7148 ZENIT EVO EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

Jalas Boot Safety 1878 Offroad, Wide fit with Aluminium Toecap & Steel Midsole, PU-Coated Leather Upper, Fleece Liner, PU Pronose, N itrile Rubber Outsole, Oil-resistant outsole, water repellant, anti-static properties, ESD, double shock absorp zones 1 * 1 PAIR

Supplier: EJENDALS

Jalas Rigger Safety 1868 King, Wide fit with Aluminium Toecap & Steel Midsole, PU-Coated Leather Upper, PU Pronose, Nitrile Rubber O utsole, Thinsulate Heat Insulation , HR Upper, Oil-resistant outsole, water repellant, anti-static properties, ESD, d 1 * 1 PAIR

Supplier: EJENDALS

ERL-4221 (3,4-EPOXYCYCLOHEXANEMETHYL 3,4-EPOXYCYCLOHEXANECARBOXYLATE) 1 * 1 PAIR

Supplier: Bioss

Transmembrane adapter protein which associates with KLRK1 to form an activation receptor KLRK1-HCST in lymphoid and myeloid cells; this receptor plays a major role in triggering cytotoxicity against target cells expressing cell surface ligands such as MHC class I chain-related MICA and MICB, and UL16-binding proteins (ULBPs); these ligands are up-regulated by stress conditions and pathological state such as viral infection and tumor transformation. Functions as docking site for PI3-kinase PIK3R1 and GRB2. Interaction of ULBPs with KLRK1-HCST triggers calcium mobilisation and activation of the PIK3R1, MAP2K/ERK, and JAK2/STAT5 signaling pathways. Both PIK3R1 and GRB2 are required for full KLRK1-HCST-mediated activation and ultimate killing of target cells. In NK cells, KLRK1-HCST signaling directly induces cytotoxicity and enhances cytokine production initiated via DAP12/TYROBP-associated receptors. In T-cells, it provides primarily costimulation for TCR-induced signals. KLRK1-HCST receptor plays a role in immune surveillance against tumors and is required for cytolysis of tumors cells; indeed, melanoma cells that do not express KLRK1 ligands escape from immune surveillance mediated by NK cells.

Supplier: Bioss

Transmembrane adapter protein which associates with KLRK1 to form an activation receptor KLRK1-HCST in lymphoid and myeloid cells; this receptor plays a major role in triggering cytotoxicity against target cells expressing cell surface ligands such as MHC class I chain-related MICA and MICB, and UL16-binding proteins (ULBPs); these ligands are up-regulated by stress conditions and pathological state such as viral infection and tumor transformation. Functions as docking site for PI3-kinase PIK3R1 and GRB2. Interaction of ULBPs with KLRK1-HCST triggers calcium mobilisation and activation of the PIK3R1, MAP2K/ERK, and JAK2/STAT5 signaling pathways. Both PIK3R1 and GRB2 are required for full KLRK1-HCST-mediated activation and ultimate killing of target cells. In NK cells, KLRK1-HCST signaling directly induces cytotoxicity and enhances cytokine production initiated via DAP12/TYROBP-associated receptors. In T-cells, it provides primarily costimulation for TCR-induced signals. KLRK1-HCST receptor plays a role in immune surveillance against tumors and is required for cytolysis of tumors cells; indeed, melanoma cells that do not express KLRK1 ligands escape from immune surveillance mediated by NK cells.

Supplier: EJENDALS

SHOE JALAS 5608 TEMPUS EU48/UK13 1 * 1 PAIR

Supplier: Avantor Fluid Handling

Complete pump systems for accurate fluid delivery - with an intuitive touch-screen interface.

Supplier: EJENDALS

SHOE JALAS 5372 SPOC EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5382 SPOC O1 EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5392 SPOC O1 EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE TEMPUS 5618 S1 P SRC EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5492 SPOC O1 EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5362 SPOC O1 EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS STREET 3065 EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5462 SPOC O1 ESD EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 5302 SPOC EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

INSOLE JALAS FX2 WINTER EU48/UK13 1 * 1 PAIR

Supplier: Ahlstrom-Munksjö

Ahlstrom-Munksjö’s qualitative grades of filter paper are recommended for use in analytical methods which determine or identify particulate constituents of a mixture irrespective of the amount present. Qualitative filter papers are often used in routine separation work that still requires high purity and consistent performance. These filter papers covers a wide range of laboratory applications, as liquids clarification, qualitative analytical separations for precipitates and buffers filtration, and are also used for soil analysis and in food and beverage testing. Wet-strengthened grades contain a reinforcement agent that increases their resistance to rupture when wet, such as in vacuum filtration, and their resistance when filtering acidic solutions. High Purity hardened filter papers grades 1288 to 1291 are also produced from cellulose with an alpha content of nearly 100% resulting in an ash content of <0,1%.

Supplier: EJENDALS

SHOE JALAS 7128 ZENIT EVO EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 2108 VIP SAFETY EU48/UK13 1 * 1 PAIR

Supplier: EJENDALS

SHOE JALAS 1550 GRAN PREMIO EU48/UK13 1 * 1 PAIR

Supplier: Bioss

Transmembrane adapter protein which associates with KLRK1 to form an activation receptor KLRK1-HCST in lymphoid and myeloid cells; this receptor plays a major role in triggering cytotoxicity against target cells expressing cell surface ligands such as MHC class I chain-related MICA and MICB, and UL16-binding proteins (ULBPs); these ligands are up-regulated by stress conditions and pathological state such as viral infection and tumor transformation. Functions as docking site for PI3-kinase PIK3R1 and GRB2. Interaction of ULBPs with KLRK1-HCST triggers calcium mobilisation and activation of the PIK3R1, MAP2K/ERK, and JAK2/STAT5 signaling pathways. Both PIK3R1 and GRB2 are required for full KLRK1-HCST-mediated activation and ultimate killing of target cells. In NK cells, KLRK1-HCST signaling directly induces cytotoxicity and enhances cytokine production initiated via DAP12/TYROBP-associated receptors. In T-cells, it provides primarily costimulation for TCR-induced signals. KLRK1-HCST receptor plays a role in immune surveillance against tumors and is required for cytolysis of tumors cells; indeed, melanoma cells that do not express KLRK1 ligands escape from immune surveillance mediated by NK cells.

Supplier: Bioss

Transmembrane adapter protein which associates with KLRK1 to form an activation receptor KLRK1-HCST in lymphoid and myeloid cells; this receptor plays a major role in triggering cytotoxicity against target cells expressing cell surface ligands such as MHC class I chain-related MICA and MICB, and UL16-binding proteins (ULBPs); these ligands are up-regulated by stress conditions and pathological state such as viral infection and tumor transformation. Functions as docking site for PI3-kinase PIK3R1 and GRB2. Interaction of ULBPs with KLRK1-HCST triggers calcium mobilisation and activation of the PIK3R1, MAP2K/ERK, and JAK2/STAT5 signaling pathways. Both PIK3R1 and GRB2 are required for full KLRK1-HCST-mediated activation and ultimate killing of target cells. In NK cells, KLRK1-HCST signaling directly induces cytotoxicity and enhances cytokine production initiated via DAP12/TYROBP-associated receptors. In T-cells, it provides primarily costimulation for TCR-induced signals. KLRK1-HCST receptor plays a role in immune surveillance against tumors and is required for cytolysis of tumors cells; indeed, melanoma cells that do not express KLRK1 ligands escape from immune surveillance mediated by NK cells.