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15167 Results for: "3-Deoxy-3-fluoro-D-glucose"

Anti-H6PD Mouse Monoclonal Antibody [clone: OTI2C12]

Anti-H6PD Mouse Monoclonal Antibody [clone: OTI2C12]

Supplier: OriGene

Anti-H6PD Mouse Monoclonal Antibody [clone: OTI2C12]

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Supelco® TraceCERT® Organic Reference Standard, Isoxaben

Supplier: Merck

It is intended to be used as a certified reference material (CRM) for calibration in chromatography and other analytical techniques. Isoxaben CRM may also find its use as described below:

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Human recombinant irisin (active)

Supplier: ENZO LIFE SCIENCES

Irisin is a hormone that is involved in converting white adipose tissue into brown adipose tissue after physical exercise.

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Anti-SGLT2 Rabbit Polyclonal Antibody

Anti-SGLT2 Rabbit Polyclonal Antibody

Supplier: Abcam

Rabbit polyclonal to SGLT2.

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Anti-SGLT2 Rabbit Polyclonal Antibody

Supplier: Abcam

Rabbit polyclonal to SGLT2.

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Anti-SGLT1 Rabbit Polyclonal Antibody

Anti-SGLT1 Rabbit Polyclonal Antibody

Supplier: Abcam

Rabbit polyclonal to SGLT1.

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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 555)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 647)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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D-(+)-Mannose ≥98%, white crystalline powder

Supplier: MP Biomedicals

Mannose is a sugar monomer of the aldohexose series of carbohydrates.

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2-(4-Fluorophenyl)ethanol ≥97%

Supplier: Thermo Fisher Scientific

2-(4-Fluorophenyl)ethanol ≥97%

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Anti-UGCGL1 Rabbit Polyclonal Antibody

Anti-UGCGL1 Rabbit Polyclonal Antibody

Supplier: Abnova

Rabbit polyclonal antibody raised against recombinant UGCGL1.

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Anti-HSPA5 Rabbit Polyclonal Antibody (FITC (Fluorescein))

Supplier: Biorbyt

Anti-HSPA5 Rabbit Polyclonal Antibody (FITC (Fluorescein))

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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 350)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 488)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PDH Rabbit Polyclonal Antibody (Cy3®)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-HSPA5 Rabbit Polyclonal Antibody Atto Dyes and Enzyme-Protein Labels

Anti-HSPA5 Rabbit Polyclonal Antibody Atto Dyes and Enzyme-Protein Labels

Supplier: Biorbyt

Anti-HSPA5 Rabbit Polyclonal Antibody Atto Dyes and Enzyme-Protein Labels

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Anti-G6PD Goat Polyclonal Antibody

Anti-G6PD Goat Polyclonal Antibody

Supplier: ProSci Inc.

Anti-G6PD Goat Polyclonal Antibody

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Anti-G6PDH Rabbit Polyclonal Antibody

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PDH Rabbit Polyclonal Antibody (FITC (Fluorescein Isothiocyanate))

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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4-Fluorobenzyl mercaptan 96%

Supplier: Thermo Fisher Scientific

4-Fluorobenzyl mercaptan 96%

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Anti-G6PDH Rabbit Polyclonal Antibody (Cy5®)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PD Mouse Monoclonal Antibody [clone: AT2F6]

Anti-G6PD Mouse Monoclonal Antibody [clone: AT2F6]

Supplier: Abnova

Mouse monoclonal antibody raised aganist partial recombinant G6PD.

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Rat CNTF ELISA kit

Rat CNTF ELISA kit

Supplier: Biorbyt

Rat CNTF ELISA Kit is used for detection of rat CNTF in cell culture supernates, serum and plasma (heparin, EDTA, citrate).

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Anti-SLC2A2 Rabbit Polyclonal Antibody

Anti-SLC2A2 Rabbit Polyclonal Antibody

Supplier: ProSci Inc.

For WB starting dilution is: 1:1000. For FACS starting dilution is: 1:10~50.

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Anti-GPI Goat Polyclonal Antibody

Anti-GPI Goat Polyclonal Antibody

Supplier: ProSci Inc.

Anti-GPI Goat Polyclonal Antibody

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Anti-SLC5A9 Rabbit Polyclonal Antibody

Anti-SLC5A9 Rabbit Polyclonal Antibody

Supplier: ProSci Inc.

Anti-SLC5A9 Rabbit Polyclonal Antibody

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Anti-G6PDH Rabbit Polyclonal Antibody (Cy5.5®)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Anti-G6PDH Rabbit Polyclonal Antibody (Cy7®)

Supplier: Bioss

Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.

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Dexamethasone 96%

Dexamethasone 96%

Supplier: Thermo Fisher Scientific

Dexamethasone 96%

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Anti-SGLT1 Rabbit Polyclonal Antibody

Anti-SGLT1 Rabbit Polyclonal Antibody

Supplier: Abcam

Rabbit polyclonal to SGLT1.

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