15167 Results for: "3-Deoxy-3-fluoro-D-glucose"
Anti-H6PD Mouse Monoclonal Antibody [clone: OTI2C12]
Supplier: OriGene
Anti-H6PD Mouse Monoclonal Antibody [clone: OTI2C12]
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Supelco® TraceCERT® Organic Reference Standard, Isoxaben
Supplier: Merck
It is intended to be used as a certified reference material (CRM) for calibration in chromatography and other analytical techniques. Isoxaben CRM may also find its use as described below:
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Human recombinant irisin (active)
Supplier: ENZO LIFE SCIENCES
Irisin is a hormone that is involved in converting white adipose tissue into brown adipose tissue after physical exercise.
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Anti-SGLT2 Rabbit Polyclonal Antibody
Supplier: Abcam
Rabbit polyclonal to SGLT2.
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Anti-SGLT2 Rabbit Polyclonal Antibody
Supplier: Abcam
Rabbit polyclonal to SGLT2.
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Anti-SGLT1 Rabbit Polyclonal Antibody
Supplier: Abcam
Rabbit polyclonal to SGLT1.
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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 555)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 647)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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D-(+)-Mannose ≥98%, white crystalline powder
Supplier: MP Biomedicals
Mannose is a sugar monomer of the aldohexose series of carbohydrates.
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2-(4-Fluorophenyl)ethanol ≥97%
Supplier: Thermo Fisher Scientific
2-(4-Fluorophenyl)ethanol ≥97%
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Anti-UGCGL1 Rabbit Polyclonal Antibody
Supplier: Abnova
Rabbit polyclonal antibody raised against recombinant UGCGL1.
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Anti-HSPA5 Rabbit Polyclonal Antibody (FITC (Fluorescein))
Supplier: Biorbyt
Anti-HSPA5 Rabbit Polyclonal Antibody (FITC (Fluorescein))
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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 350)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PDH Rabbit Polyclonal Antibody (Alexa Fluor® 488)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PDH Rabbit Polyclonal Antibody (Cy3®)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-HSPA5 Rabbit Polyclonal Antibody Atto Dyes and Enzyme-Protein Labels
Supplier: Biorbyt
Anti-HSPA5 Rabbit Polyclonal Antibody Atto Dyes and Enzyme-Protein Labels
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Anti-G6PD Goat Polyclonal Antibody
Supplier: ProSci Inc.
Anti-G6PD Goat Polyclonal Antibody
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Anti-G6PDH Rabbit Polyclonal Antibody
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PDH Rabbit Polyclonal Antibody (FITC (Fluorescein Isothiocyanate))
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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4-Fluorobenzyl mercaptan 96%
Supplier: Thermo Fisher Scientific
4-Fluorobenzyl mercaptan 96%
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Anti-G6PDH Rabbit Polyclonal Antibody (Cy5®)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PD Mouse Monoclonal Antibody [clone: AT2F6]
Supplier: Abnova
Mouse monoclonal antibody raised aganist partial recombinant G6PD.
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Rat CNTF ELISA kit
Supplier: Biorbyt
Rat CNTF ELISA Kit is used for detection of rat CNTF in cell culture supernates, serum and plasma (heparin, EDTA, citrate).
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Anti-SLC2A2 Rabbit Polyclonal Antibody
Supplier: ProSci Inc.
For WB starting dilution is: 1:1000. For FACS starting dilution is: 1:10~50.
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Anti-GPI Goat Polyclonal Antibody
Supplier: ProSci Inc.
Anti-GPI Goat Polyclonal Antibody
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Anti-SLC5A9 Rabbit Polyclonal Antibody
Supplier: ProSci Inc.
Anti-SLC5A9 Rabbit Polyclonal Antibody
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Anti-G6PDH Rabbit Polyclonal Antibody (Cy5.5®)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-G6PDH Rabbit Polyclonal Antibody (Cy7®)
Supplier: Bioss
Defects in G6PD are the cause of chronic non-spherocytic hemolytic anemia (CNSHA) . Deficiency of G6PD is associated with hemolytic anemia in two different situations. First, in areas in which malaria has been endemic, G6PD-deficiency alleles have reached high frequencies (1% to 50%) and deficient individuals, though essentially asymptomatic in the steady state, have a high risk of acute hemolytic attacks. Secondly, sporadic cases of G6PD deficiency occur at a very low frequencies, and they usually present a more severe phenotype. Several types of CNSHA are recognized. Class-I variants are associated with severe NSHA; class-II have an activity <10% of normal; class-III have an activity of 10% to 60% of normal; class-IV have near normal activity.
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Anti-SGLT1 Rabbit Polyclonal Antibody
Supplier: Abcam
Rabbit polyclonal to SGLT1.