20632 Results for: "Acetylthio-PEG3-C2-Azide&amp"

Supplier: Abcam

Rabbit monoclonal [EPR19993-105] to Niemann Pick C2 - BSA and Azide free (Capture).

Supplier: US Biological

Anti-C2 Rabbit Polyclonal Antibody (AP (Alkaline Phosphatase))

Supplier: ProSci Inc.

Component C2 is part of the classical pathway of complement system. Activated C1 cleaves C2 into C2a and C2b. C2a leads to activation of C3. Deficiency of C2 has been reported to associated with certain autoimmune diseases.Component C2 is part of the classical pathway of complement system. Activated C1 cleaves C2 into C2a and C2b. C2a leads to activation of C3. Deficiency of C2 has been reported to associated with certain autoimmune diseases.

Supplier: ProSci Inc.

Anti-C2 Rabbit Polyclonal Antibody

Supplier: Abcam

Rabbit monoclonal [EPR19993-63] to Niemann Pick C2 - BSA and Azide free (Detector).

Supplier: Abnova

Anti-C2 Rabbit Polyclonal Antibody

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: OriGene

Anti-C2 Rabbit Polyclonal Antibody

Supplier: US Biological

Anti-CRX Mouse Monoclonal Antibody [clone: F6-C2]

Supplier: US Biological

Anti-Complement C2 Mouse Monoclonal Antibody [clone: 6F61]

Supplier: ProSci Inc.

Component C2 is part of the classical pathway of complement system. Activated C1 cleaves C2 into C2a and C2b. C2a leads to activation of C3. Deficiency of C2 has been reported to associated with certain autoimmune diseases.Component C2 is part of the classical pathway of complement system. Activated C1 cleaves C2 into C2a and C2b. C2a leads to activation of C3. Deficiency of C2 has been reported to associated with certain autoimmune diseases.Component C2 is part of the classical pathway of complement system. Activated C1 cleaves C2 into C2a and C2b. C2a leads to activation of C3. Deficiency of C2 has been reported to associated with certain autoimmune diseases. Publication Note: This RefSeq record includes a subset of the publications that are available for this gene. Please see the Entrez Gene record to access additional publications.

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: Abcam

Rabbit monoclonal [EPNCIR152] to hnRNP C1/C2.

Supplier: Abcam

Rabbit monoclonal [EP3034Y] to hnRNP C1/C2.

Supplier: Abcam

Rabbit polyclonal to C2.

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: QED Bioscience

Anti-Cyclic AMP Sheep Polyclonal Antibody

Supplier: US Biological

Anti-C2 Rabbit Polyclonal Antibody (PE (Phycoerythrin))

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: Bioss

Component C2 is a serum glycoprotein that functions as part of the classical pathway of the complement system. Activated C1 cleaves C2 into C2a and C2b. The serine proteinase C2a then combines with complement factor 4b to create the C3 or C5 convertase. Deficiency of C2 has been reported to associated with certain autoimmune diseases and SNPs in this gene have been associated with altered susceptibility to age-related macular degeneration. This gene localizes within the class III region of the MHC on the short arm of chromosome 6. Alternative splicing results in multiple transcript variants encoding distinct isoforms. Additional transcript variants have been described in publications but their full-length sequence has not been determined.[provided by RefSeq, Mar 2009].

Supplier: Abcam

Rabbit monoclonal [EPR10746(B)] to AMPS.

Supplier: Abnova

Mouse polyclonal antibody raised against a full-length human C2 protein.

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: ANTIBODIES.COM

Human Complement C2 ELISA kit is a sandwich Enzyme-Linked Immunosorbent Assay (sELISA) designed for the in vitro quantitative determination of human Complement C2 in serum, plasma, tissue homogenates, and other biological fluids.

Supplier: US Biological

Anti-NMDA 1 Receptor, C2’ Splice Variant Rabbit Polyclonal Antibody

Supplier: ANTIBODIES.COM

Anti-PEG3 Rabbit Polyclonal Antibody

Supplier: Abnova

Mouse polyclonal antibody raised against a full-length human C2 protein.

Supplier: Bioss

Induces apoptosis in cooperation with SIAH1A. Acts as a mediator between p53/TP53 and BAX in a neuronal death pathway that is activated by DNA damage. Acts synergistically with TRAF2 and inhibits TNF induced apoptosis through activation of NF-kappa-B (By similarity). Possesses a tumor suppressing activity in glioma cells.

Supplier: Cayman Chemical

For direct quantification of cAMP from a variety of biological samples.

Supplier: Abcam

Rabbit monoclonal [EPR10747(B)] to AMPS.