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451 results for "TS6ASV9-1P"

451 Results for: "TS6ASV9-1P"

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Papír pro separaci fází, grade 1PS, Whatman™

Papír pro separaci fází, grade 1PS, Whatman™

Supplier: Whatman products (Cytiva)

1PS phase-separating paper is a hydrophobic filter for routine solvent extraction. Within seconds it retains aqueous phases, while solvent phases flow through.

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Battery-Powered Digital Flow Rate Monitor and Totalizer 43643

Battery-Powered Digital Flow Rate Monitor and Totalizer 43643

Supplier: Avantor Fluid Handling

Use in areas where AC power is not readily available.

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Anti-PLB Rabbit Polyclonal Antibody (Cy5.5®)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Cy5®)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Alexa Fluor® 647)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Cy3®)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Alexa Fluor® 488)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Cy7®)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Alexa Fluor® 350)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (FITC (Fluorescein Isothiocyanate))

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Ser16 Rabbit Polyclonal Antibody (Alexa Fluor® 750)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterised by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterised by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Ser16 Rabbit Polyclonal Antibody (Alexa Fluor® 680)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterised by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterised by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-ALG11 Rabbit Polyclonal Antibody

Supplier: Bioss

Mannosyltransferase involved in the last steps of the synthesis of Man5GlcNAc(2)-PP-dolichol core oligosaccharide on the cytoplasmic face of the endoplasmic reticulum. Catalyzes the addition of the 4th and 5th mannose residues to the dolichol-linked oligosaccharide chain.Involvement in disease:Defects in ALG11 are the cause of congenital disorder of glycosylation type 1P (CDG1P). A multisystem disorder caused by a defect in glycoprotein biosynthesis and characterized by under-glycosylated serum glycoproteins. Congenital disorders of glycosylation result in a wide variety of clinical features, such as defects in the nervous system development, psychomotor retardation, dysmorphic features, hypotonia, coagulation disorders, and immunodeficiency. The broad spectrum of features reflects the critical role of N-glycoproteins during embryonic development, differentiation, and maintenance of cell functions.

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Anti-PLB Rabbit Polyclonal Antibody (HRP (Horseradish Peroxidase))

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-PLB Rabbit Polyclonal Antibody (Alexa Fluor® 555)

Supplier: Bioss

Defects in PLN are the cause of cardiomyopathy dilated type 1P (CMD1P) . Dilated cardiomyopathy is a disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.Defects in PLN are the cause of cardiomyopathy familial hypertrophic type 18 (CMH18) . CMH18 is a hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

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Anti-Presenilin 1/PS-1 Rabbit Monoclonal Antibody [clone: EP2000Y]

Supplier: Abcam

Anti-Presenilin 1/PS-1 Rabbit Monoclonal Antibody [clone: EP2000Y]

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[EN]CAPTAIR 633 SMART 1P D R I EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 633 SMART 1P D R I EU 1 * 1 KS

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[EN]CAPTAIR 483 SMART 1P D R I EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 483 SMART 1P D R I EU 1 * 1 KS

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[EN]HALO 25 SMART 1P CP CTHR W EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]HALO 25 SMART 1P CP CTHR W EU 1 * 1 KS

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[EN]CAPTAIR 714 SMART 1P P VD EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 714 SMART 1P P VD EU 1 * 1 KS

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[EN]CAPTAIR 714 SMART 1P D VG EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 714 SMART 1P D VG EU 1 * 1 KS

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[EN]CAPTAIR 633 SMART 1P P VD EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 633 SMART 1P P VD EU 1 * 1 KS

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[EN]CAPTAIR 633 SMART 1P P RG EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 633 SMART 1P P RG EU 1 * 1 KS

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[EN]CAPTAIR 392 SMART 1P P RG EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 392 SMART 1P P RG EU 1 * 1 KS

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[EN]CAPTAIR 481 SMART 1P D VD EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 481 SMART 1P D VD EU 1 * 1 KS

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[EN]CAPTAIR 714 SMART 1P D R T EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 714 SMART 1P D R T EU 1 * 1 KS

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[EN]CAPTAIR 633 SMART 1P D R T EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 633 SMART 1P D R T EU 1 * 1 KS

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[EN]HANDLE FOR FURNACE RG-1P.120.84 1 * 1 KS

Supplier: NABERTHERM

[EN]HANDLE FOR FURNACE RG-1P.120.84 1 * 1 KS

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[EN]CAPTAIR 633 SMART 1P D VS I EU 1 * 1 KS

Supplier: ERLAB CAPTAIRE

[EN]CAPTAIR 633 SMART 1P D VS I EU 1 * 1 KS

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